Polypeptide Hormone 10mg/vail Melanotan 1

Polypeptide Hormone 10mg/vail Melanotan 1

Polypeptide Hormone Melanotan 1 COA: Description MT-1 (or Melanotan I,or Afamelanotide,or CUV-1647) is a synthetic analog of peptides nature of endogenous melanocortine peptide alpha-melanocyte stimulating hormone.It has hormones nature as well as common proteins features,its molecular...

Product Details

Polypeptide Hormone 10mg/vail Melanotan 1


COA


AppearanceWhite to off white powderConsistent
Purity(HPLC)≥98%99.10%
Water<6.0%4.40%
Acetic Acid3.0-8.0%6.85%
Trifluoroacetic Acid<0.25%0.18%
Peptide≥80%84.56%
MSConsistentConsistent


Description


MT-1 (or Melanotan I,or Afamelanotide,or CUV-1647) is a synthetic analog of peptides nature of endogenous melanocortine peptide alpha-melanocyte stimulating hormone.It has hormones nature as well as common proteins features,its molecular weight/mass is 1646.88 and its molecular formula is C78H111N21O19.


Developed research compound is quite stable cyclic form of original hormone but specifically has much longer half-life period.It works as agonist of specific melanocortin receptor and possesses such property as being selective;And thus it promotes increased level of eumelanin above usual pheomelanin,which provides higher efficacy of photoprotection abilities.


Melanotan-1 Effects


The Melanotan-1, known as Afamelanotide, is a synthetic analog of the naturally occurring melanocortin peptide hormone alpha-melanocyte stimulating hormone (α-MSH), and α-MSH has been shown to induce skin pigmentation through melanogenesis and thereby subsequently reduce sun (UV) damage to UV exposed skin in preliminary studies and clinical trials.


Afamelanotide is a synthetic peptide analogue of the naturally occurring alpha-melanocyte stimulating hormone (a-MSH) with potential photoprotective activity. Mimicking the action of a-MSH, afamelanotide stimulates melanocytes to increase the production and release of melanin. Increased melanocyte melanin may protect against ultraviolet radiation (UVR)-initiated cellular DNA damage, oxidation of membrane proteins, and alterations in intracellular signaling processes in epidermal cells. Endogenously, a-MSH is released by skin cells in response to UVR exposure, stimulating melanocytes to produce and release melanin.


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